Formulation aidBuffer and chelatorAHA by class

Citric Acid

Citric Acid · INCI Citric Acid

Citric acid is one of the most widely used ingredients in food, pharmacy and cosmetics, and it belongs to the alpha hydroxy acid family. That family membership is where most of the confusion starts. In a product held at the acidity of skin, it is not doing the work an AHA is famous for. It is setting the pH and binding metals, which are duller sentences and more useful jobs.

An alpha hydroxy acid by classification, working here as a pH ingredient rather than as an exfoliant.

Citric acid, the colourless crystalline powder, odourless and water-soluble, and nothing else.
Citric acid, the colourless crystalline powder, odourless and water-soluble, and nothing else.

What it is

Citric acid is a small organic acid with three acid groups on it, and those three groups are most of why it is useful. The commercial material is not squeezed out of fruit. It is made by microbial fermentation, classically with the mould Aspergillus niger growing on a sugar substrate, a route industrialised somewhere around 1916 to 1919 and largely unchanged in principle since. What arrives is a colourless, odourless crystalline powder that dissolves readily in water.

Three acid groups means three points at which the molecule gives up or takes back a proton, and that is what makes it a buffer rather than simply an acid. It can push a formula down to a chosen acidity and then hold its ground when something tries to push it back. Put it with sodium citrate, which is its own salt, and the pair forms a citrate buffer: more stubborn than either alone, and gentler than the acid on its own. It also sequesters trace metal ions, iron and copper among them, which is a second and quite separate use.

The thing worth understanding about citric acid is that what it does depends almost entirely on where the pH sits. In its free acid form at low pH, like other alpha hydroxy acids, it loosens the bonds between surface skin cells and speeds their shedding. As the pH climbs towards and past its own dissociation points, more of it exists as citrate salt, and that action falls away. The exfoliating benefit and the stinging share the same lever. Our formulas sit between pH 5 and 6, which is roughly where skin sits, and at that acidity citric acid is a pH ingredient and a chelator rather than an exfoliant. The research that supports the exfoliating and structural claims was produced at 20%, or at a pH the papers never recorded.

What it does in our formulas

Two jobs, and both of them are done to the formula rather than to your skin.

01

It is the acid half of a citrate buffer

Citric acid carries three acid groups and gives up or takes back a proton at three different points along the pH scale. That is what separates a buffer from a plain acid: it can bring a formula to a chosen acidity and then resist being pushed back off it. Paired with its own salt, sodium citrate, it makes a citrate buffer, which holds a product between pH 5 and 6 far more stubbornly than either half would alone, and takes the bite off the acid while it does. The independent expert panel that reviews cosmetic ingredients lists pH adjuster as one of its two primary reported cosmetic functions.

Well characterised
02

It binds trace metals, which is reason enough on its own

Iron and copper get into any formula, from the water and from the raw materials, and metal ions are what set oils oxidising. Citric acid sequesters them, and chelation is the second function that same panel lists for it. That protects the oxidation-prone materials in a formula, and it supports organic-acid preservation, which only works in acid conditions. Neither of those is a claim about skin, and neither needs to be. A formula that stays chemically quiet is the whole point of the ingredient.

Well characterised
A modest bonus

One quieter point, at its real weight. Skin's own surface is mildly acidic, and the research shows that acidity supports the enzymes that build the barrier's lipids, restrains the enzymes that break the barrier down, and tips the surface balance towards the microbes that prefer acid. An acid that holds a product in that band is working with all three rather than against them. The direct human evidence is thinner than the mechanism, though. The study that sits closest to our pH used cotton textiles treated with citric acid, worn against the skin for two weeks, and it lowered surface pH and improved barrier measures. That is a treated fabric rather than a cream, which is why this sits here as a supporting note and not as a claim.

What the research does not show

Citric acid is an alpha hydroxy acid, and that single fact carries an enormous amount of marketing behind it. Much of the science underneath is real. It was just produced under conditions that have little to do with a leave-on product at the acidity of skin: a different concentration, a different pH, or a different material altogether.

Marketed as A collagen-building acid
There is real research behind this one, and it is worth reading properly rather than dismissing. A 1997 study applied a 20% citric acid lotion to sun-damaged skin for three months and found, on biopsy, increased viable epidermal thickness and more dermal glycosaminoglycan. The condition that matters is the 20%: that is several times any normal leave-on level, the paper did not report the pH it was run at, and there is nothing to say the finding survives a fraction of a percent at the acidity of skin. The precise figures that travel with this study, a 40% increase in viable epidermis and a 16.3% increase in skin-fold thickness, are not in the primary paper, which reports direction of change only, and a widely repeated 25% increase in skin thickness traces to a patent rather than to a trial.
Marketed as Inhibits tyrosinase
The named mechanism is the wrong one. In cell culture, excess citric acid did suppress melanin production in human melanocytes, but through the MITF and GSK3-beta/beta-catenin signalling pathway, not through tyrosinase. That is a laboratory observation at concentrations that do not map onto a leave-on product, and it is worth adding that the same molecule promoted melanin synthesis in a mouse melanoma line in the same study. The direction of the effect depends on the cell.
Marketed as An antimicrobial active
The concentration figures quoted for this, minimum inhibitory concentrations against Propionibacterium acnes, are measurements of lime juice, a whole-fruit extract with a great deal in it besides citric acid. The pattern repeats: antimicrobial data attributed to citric acid elsewhere turn out to describe citrate-based polymers, and a soothing claim for reactive skin turns out to rest on citron essential oil. None of those materials is the crystalline acid in a formula. Whatever effect citric acid has on the surface is mediated by pH, which is a quieter and better-supported story.

None of this is an argument against the ingredient. It is an argument about conditions. Change the pH and raise the concentration and citric acid does become an exfoliating acid, with everything that comes with that, including the sting and the caution about sunlight. Held where we hold it, it is doing quieter chemistry, and that is the version of it described on this page.

On the label and in the range

Citric acid does not appear on a Biotaderm ingredients list, because it is not added as a formula ingredient. It is used during manufacture, in very small quantities, to bring each batch to its working pH.

Its salt, sodium citrate, is a formula ingredient, and it sits high in every list at a published percentage. The difference between the two is worth understanding. Reaching an acidity takes almost nothing. Holding it steady across a shelf life takes a real amount, and that is the salt's job rather than the acid's.

Is it suitable for me?

The independent expert panel that reviews cosmetic ingredients assessed citric acid and its salts and found them safe as used, and repeated-insult patch testing at 4% produced no irritation and no sensitisation. True contact allergy to citric acid is uncommon. It suits a vegan routine, and UK and EU cosmetic law places no concentration restriction on it. You will sometimes see 4% quoted as its legal ceiling, which it is not: that figure is the highest leave-on level the panel found in use when it surveyed the market, a description rather than a cap, and the same panel noted that citric acid is chemically and biologically distinct from the alpha hydroxy acids that do carry recommended limits. What genuinely governs tolerance here is concentration and pH together. Severe facial burns have been reported from a 10% peel, and one comparative study found citric acid more irritating than lactic acid on sensitive skin. At buffer levels in a product held between pH 5 and 6 that risk is low, and the same chemistry that makes it mild at low dose is what makes it sharp at high dose and low pH.

Vegan-friendlySensitive-skin suitable

As with anything new, if your skin reacts easily it is worth patch testing first.

Formulation Clear
Formulation Clear
Science led
Science led
Cruelty free
Cruelty free

The research behind this page

We review the published literature. We do not conduct the studies below.

  1. Fiume MM, Heldreth BA, Bergfeld WF, et al. Safety Assessment of Citric Acid, Inorganic Citrate Salts, and Alkyl Citrate Esters as Used in Cosmetics. Int J Toxicol 2014;33(2 Suppl):16S-46S.
  2. Tang S-C, Yang J-H. Dual Effects of Alpha-Hydroxy Acids on the Skin. Molecules 2018;23(4):863.
  3. Schmid-Wendtner MH, Korting HC. The pH of the skin surface and its impact on the barrier function. Skin Pharmacol Physiol 2006;19(6):296-302.
  4. Jaeger T, Rothmaier M, Zander H, Ring J, Gutermuth J, Anliker MD. Acid-coated Textiles (pH 5.5-6.5), a New Therapeutic Strategy for Atopic Eczema? Acta Derm Venereol 2015;95(6):659-663.
  5. Bernstein EF, Underhill CB, Lakkakorpi J, Ditre CM, Uitto J, Yu RJ, Scott EV. Citric acid increases viable epidermal thickness and glycosaminoglycan content of sun-damaged skin. Dermatol Surg 1997;23(8):689-694.
  6. Zhou S, Sakamoto K. Citric acid promoted melanin synthesis in B16F10 mouse melanoma cells, but inhibited it in human epidermal melanocytes and HMV-II melanoma cells via the GSK3-beta/beta-catenin signalling pathway. PLOS ONE 2020;15(12):e0243565.
Used in manufacture to set batch pH. Not listed as an ingredient in any product. Shop the range